Phys. Rev. -- Table of Contents (April 2009, 89, (2))

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Contents: Volume 89, Issue 2; April, 2009. [Index by Author] [Editorial Board]

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Articles

Michael J. Tisdale: Mechanisms of Cancer Cachexia
Physiol. Rev. 89: 381-410, 2009; doi:10.1152/physrev.00016.2008 [Abstract] [Full Text] [PDF]

Shuiping Dai, Duane D. Hall, and Johannes W. Hell: Supramolecular Assemblies and Localized Regulation of Voltage-Gated Ion Channels
Physiol. Rev. 89: 411-452, 2009; doi:10.1152/physrev.00029.2007 [Abstract] [Full Text] [PDF]

Riitta Hari and Miiamaaria V. Kujala: Brain Basis of Human Social Interaction: From Concepts to Brain Imaging
Physiol. Rev. 89: 453-479, 2009; doi:10.1152/physrev.00041.2007 [Abstract] [Full Text] [PDF]

J.-L. Balligand, O. Feron, and C. Dessy: eNOS Activation by Physical Forces: From Short-Term Regulation of Contraction to Chronic Remodeling of Cardiovascular Tissues
Physiol. Rev. 89: 481-534, 2009; doi:10.1152/physrev.00042.2007 [Abstract] [Full Text] [PDF]

Richard Kvetnansky, Esther L. Sabban, and Miklos Palkovits: Catecholaminergic Systems in Stress: Structural and Molecular Genetic Approaches
Physiol. Rev. 89: 535-606, 2009; doi:10.1152/physrev.00042.2006 [Abstract] [Full Text] [PDF]

Carmen Ruiz de Almodovar, Diether Lambrechts, Massimiliano Mazzone, and Peter Carmeliet: Role and Therapeutic Potential of VEGF in the Nervous System
Physiol. Rev. 89: 607-648, 2009; doi:10.1152/physrev.00031.2008 [Abstract] [Full Text] [PDF]

Rainer Spanagel: Alcoholism: A Systems Approach From Molecular Physiology to Addictive Behavior
Physiol. Rev. 89: 649-705, 2009; doi:10.1152/physrev.00013.2008 [Abstract] [Full Text] [PDF]

Jürgen Sandkühler: Models and Mechanisms of Hyperalgesia and Allodynia
Physiol. Rev. 89: 707-758, 2009; doi:10.1152/physrev.00025.2008 [Abstract] [Full Text] [PDF]

Cover

Cover: Although originally described as a key angiogenic factor, vascular endothelial growth factor (VEGF) plays also a crucial role in the nervous system. It is now well established that VEGF induces proliferation, survival, migration, and differentiation of distinct neuronal cell types. Moreover, VEGF has a therapeutic potential in several neurological diseases. For example, when VEGF is administered to amyotrophic lateral sclerosis (ALS) animal models, it protects motor neurons from degeneration, improves motor performance, and prolongs survival. The illustration depicts the healthier status of a motor neuron receiving VEGF (in yellow) compared with a dying motor neuron that does not receive VEGF. See Ruiz de Almodovar, Carmen, Diether Lambrechts, Massimiliano Mazzone, and Peter Carmeliet. Physiol Rev 89: 607-648, 2009.