Phys. Rev. -- Table of Contents (July 2007, 87, (3))

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[Cover Caption]
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Contents: Volume 87, Issue 3; July, 2007. [Index by Author] [Editorial Board]

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Articles

Mark F. Mehler and John S. Mattick: Noncoding RNAs and RNA Editing in Brain Development, Functional Diversification, and Neurological Disease
Physiol. Rev. 87: 799-823, 2007; doi:10.1152/physrev.00036.2006 [Abstract] [Full Text] [PDF]

Mark Donowitz and Xuhang Li: Regulatory Binding Partners and Complexes of NHE3
Physiol. Rev. 87: 825-872, 2007; doi:10.1152/physrev.00030.2006 [Abstract] [Full Text] [PDF]

Bruce S. McEwen: Physiology and Neurobiology of Stress and Adaptation: Central Role of the Brain
Physiol. Rev. 87: 873-904, 2007; doi:10.1152/physrev.00041.2006 [Abstract] [Full Text] [PDF]

Nina Heldring, Ashley Pike, Sandra Andersson, Jason Matthews, Guojun Cheng, Johan Hartman, Michel Tujague, Anders Ström, Eckardt Treuter, Margaret Warner, and Jan-Åke Gustafsson: Estrogen Receptors: How Do They Signal and What Are Their Targets
Physiol. Rev. 87: 905-931, 2007; doi:10.1152/physrev.00026.2006 [Abstract] [Full Text] [PDF]

Xiaoyan Zhu, Anatoli S. Gleiberman, and Michael G. Rosenfeld: Molecular Physiology of Pituitary Development: Signaling and Transcriptional Networks
Physiol. Rev. 87: 933-963, 2007; doi:10.1152/physrev.00006.2006 [Abstract] [Full Text] [PDF]

Debbie Willoughby and Dermot M. F. Cooper: Organization and Ca²⁺ Regulation of Adenylyl Cyclases in cAMP Microdomains
Physiol. Rev. 87: 965-1010, 2007; doi:10.1152/physrev.00049.2006 [Abstract] [Full Text] [PDF]

Svetlana Lutsenko, Natalie L. Barnes, Mee Y. Bartee, and Oleg Y. Dmitriev: Function and Regulation of Human Copper-Transporting ATPases
Physiol. Rev. 87: 1011-1046, 2007; doi:10.1152/physrev.00004.2006 [Abstract] [Full Text] [PDF]

Toshinori Yoshida and Rubin M. Tuder: Pathobiology of Cigarette Smoke-Induced Chronic Obstructive Pulmonary Disease
Physiol. Rev. 87: 1047-1082, 2007; doi:10.1152/physrev.00048.2006 [Abstract] [Full Text] [PDF]

Cover

Cover: Current pathogenesis of emphysema caused by cigarette smoke and air pollutants. Chronic exposure to cigarette smoke and environmental air pollutants triggers the activation of inflammatory processes involving dendritic cells (DC), CD4 and CD8 lymphocytes, neutrophils (PMNs), and alveolar macrophages, causing synthesis and release of extracellular matrix proteases. Excessive oxidants originating from cigarette smoke or air pollutants, inflammatory cells, and alveolar septal cells overcome antioxidant defenses, causing amplification of alveolar inflammation, matrix proteolysis, and alveolar apoptosis (ap). Altered cell signaling and excessive oxidative stress lead to alveolar apoptosis, which, with extracellular matrix proteases, promotes lung matrix damage with fragmentation of elastin fibers, abnormal collagen assembly, and loss of alveolar structures. See Yoshida, Toshinori, and Rubin M. Tuder. Physiol Rev 87: 1047–1082, 2007.