Phys. Rev. -- Table of Contents (April 2007, 87, (2))

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Contents: Volume 87, Issue 2; April, 2007. [Index by Author] [Editorial Board]

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Stanley Nattel, Ange Maguy, Sabrina Le Bouter, and Yung-Hsin Yeh: Arrhythmogenic Ion-Channel Remodeling in the Heart: Heart Failure, Myocardial Infarction, and Atrial Fibrillation
Physiol. Rev. 87: 425-456, 2007; doi:10.1152/physrev.00014.2006 [Abstract] [Full Text] [PDF]

Henk E. D. J. ter Keurs and Penelope A. Boyden: Calcium and Arrhythmogenesis
Physiol. Rev. 87: 457-506, 2007; doi:10.1152/physrev.00011.2006 [Abstract] [Full Text] [PDF]

David B. Savage, Kitt Falk Petersen, and Gerald I. Shulman: Disordered Lipid Metabolism and the Pathogenesis of Insulin Resistance
Physiol. Rev. 87: 507-520, 2007; doi:10.1152/physrev.00024.2006 [Abstract] [Full Text] [PDF]

Preeti Ahuja, Patima Sdek, and W. Robb MacLellan: Cardiac Myocyte Cell Cycle Control in Development, Disease, and Regeneration
Physiol. Rev. 87: 521-544, 2007; doi:10.1152/physrev.00032.2006 [Abstract] [Full Text] [PDF]

Anthony T. Blikslager, Adam J. Moeser, Jody L. Gookin, Samuel L. Jones, and Jack Odle: Restoration of Barrier Function in Injured Intestinal Mucosa
Physiol. Rev. 87: 545-564, 2007; doi:10.1152/physrev.00012.2006 [Abstract] [Full Text] [PDF]

Laerte Oliveira, Claudio M. Costa-Neto, Clovis R. Nakaie, Shirley Schreier, Suma I. Shimuta, and Antonio C. M. Paiva: The Angiotensin II AT₁ Receptor Structure-Activity Correlations in the Light of Rhodopsin Structure
Physiol. Rev. 87: 565-592, 2007; doi:10.1152/physrev.00040.2005 [Abstract] [Full Text] [PDF] ]Supplemental Material[

J. Kevin Foskett, Carl White, King-Ho Cheung, and Don-On Daniel Mak: Inositol Trisphosphate Receptor Ca²⁺ Release Channels
Physiol. Rev. 87: 593-658, 2007; doi:10.1152/physrev.00035.2006 [Abstract] [Full Text] [PDF]

Geoffrey Burnstock: Physiology and Pathophysiology of Purinergic Neurotransmission
Physiol. Rev. 87: 659-797, 2007; doi:10.1152/physrev.00043.2006 [Abstract] [Full Text] [PDF]

Cover

Cover: Mechanism of fatty acid-induced insulin resistance in the liver. Increases in diacylglycerol (DAG), due to increased lipogenesis and/or decreased mitochondrial fatty acid oxidation, activate protein kinase C (PKC)-ε, which binds to and activates the insulin receptor kinase resulting in reduced insulin-stimulated insulin receptor substrate (IRS)-1 and IRS-2 tyrosine phosphorylation. This in turn results in reduced insulin activation of phosphoinositol (PI) 3-kinase and AKT2. Reduced ATK2 activation results in lower glycogen synthase kinase-3 (GSK3) phosphorylation and lower forkhead box protein O (FOXO) phosphorylation, which in turn results in lower insulin-stimulated liver glycogen synthesis and decreased suppression of hepatic gluconeogenesis, respectively. See Savage, David B., Kitt Falk Petersen, and Gerald I. Shulman. Physiol Rev 87: 507-520, 2007.